Histone deacetylase 1 reduces NO production in endothelial cells via lysine deacetylation of NO synthase 3

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Histone deacetylase 1 reduces NO production in endothelial cells via lysine deacetylation of NO synthase 3.

The lysine acetylation state of nonhistone proteins may be regulated through histone deacetylases (HDACs). Evidence suggests that nitric oxide (NO) synthase 3 (NOS3; endothelial NOS) is posttranslationally lysine acetylated, leading to increased NO production in the endothelium. We tested the hypothesis that NOS3 is lysine acetylated and that upregulated HDAC1-mediated deacetylation leads to re...

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ژورنال

عنوان ژورنال: American Journal of Physiology-Heart and Circulatory Physiology

سال: 2014

ISSN: 0363-6135,1522-1539

DOI: 10.1152/ajpheart.00243.2014